Overview
Lazy eye, medically known as amblyopia (am-blee-OH-pee-a),
is the loss or lack of development of vision, usually in
one eye. It affects about 2 percent of children and is
rare among adults. In most cases, the abilities of one
eye are limited so it does not develop properly. The
brain gives preference to the dominant eye. If left
untreated, the brain will eventually “ignore” images
from the weaker eye, and eventually its nerve
connections degenerate.
This degenerative process usually begins with an
inherited condition and appears during infancy or early
childhood. Lazy eye needs to be diagnosed between birth
and early school age (age 8 or 9), since it is during
this period that brain “chooses” its visual pathway and
may ignore the weaker eye permanently. Even with early
treatment, ongoing complications and recurrence are
possible.
There are several underlying causes, including:
- Crossed eyes (strabismus). This causes
double vision (diplopia) when children use both eyes
at the same time. One image may appear clearer than
the other, and the brain will block out the weaker
image when double vision is prolonged.
- Focus inequality, in which one eye is
much more nearsighted, farsighted or astigmatic than
the other. Again, the brain gives preference to the
stronger eye.
- Structural problems in the eye or eyelid,
such as a cataract or ptosis (a drooping eyelid).
Symptoms
Lazy eye is not always easy to recognize, since a
child with worse vision in one eye does not necessarily
have lazy eye. This is why all children – including
those with no symptoms – should get a complete eye
examination by age 3, and sooner if there is a family
history of any eye condition or disease or if you detect
any of these symptoms:
- The ability to see well on only one side, or
clearly seeing some but not all objects clearly
- Trouble judging depth or distance
- Covering one eye to see, or tilting the head to
read or perform other activities
- Frequent rubbing or winking
- Bumping into objects on the side of the weaker
eye or other signs of clumsiness
- Infants may cry if one eye is covered
Prevention
To prevent permanent loss of vision in a "weak" eye,
newborns should receive an eye exam to rule out
congenital abnormalities. Visual acuity testing should
begin by age 3, and be done at least annually
thereafter. If the results of a screening exam suggest
that your child has lazy eye, you will be referred to an
ophthalmologist for further testing and assessment.
Treatment
Treatment is usually successful with prompt
intervention and treatment. There are three goals:
correcting the underlying problems, strengthening the
weaker eye and producing a clear visual image in both
eyes. To achieve this, your eyecare practitioner may
employ:
- Eyeglasses with prism, concave, convex or
bifocal lenses to strengthen and restore focus to
the weaker eye, encouraging the brain to use it over
time. Eyeglasses can also be used to treat focus
inequality caused by conditions such as
nearsightedness or farsightedness.
- Contact lenses are also used in cases of
severe refractive inequality (between the two eyes),
or a single contact lens may be prescribed for the
amblyopic eye.
- Monitored patching consists of covering
the stronger eye for defined periods – usually for
at least 6 months – to strengthen the muscles that
control the weaker eye. Once the child’s vision is
restored, occasional patching may still be needed
for several years. As an alternative, some doctors
use an opaque contact lens or prescribe atropine eye
drops to temporarily blur vision in the stronger
eye.
- Vision therapy, or orthoptics, can
strengthen, coordinate and improve the functions of
both eyes, especially in the early years of life.
- Surgery can “reposition” eye muscles to
restore permanent control of the weaker eye. If
cataracts are the cause of the amblyopia, they must
be surgically removed. After cataract removal, an
artificial lens can restore focus, and eventually
restore strength to the weaker eye. Other surgical
procedures may also be beneficial when other eye
diseases are the cause.
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